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Possible cause of, and treatment for, non-familial Parkinson?s

Researchers have identified a protein trafficking defect within brain cells that may underlie the common form of Parkinson's disease. The defect is at a point of convergence for the action of at least three different genes that had been implicated in prior studies of Parkinson's disease. Whereas most molecular studies focus on mutations associated with rare familial forms of the disease, these findings relate directly to the common non-familial form of Parkinson?s.
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